| The
individual lysis capacity – a natural factor in athletic
selection |
|
Based
on their ability to realise, under basal conditions, an individual
lysis capacity, individuals can be describes as being:
•
Low NK – low individual lysis capacity- people who have a
capacity of up to 25% for natural killer cells lysis;
Picture 1
Individual lysis capacity
recorded in judoka.
Legend: the ordinate shows the number of
cases recorded; the abscissa shows the values for:
- low NK
< 25%,
- middle NK 25% - 29% and 30%-39%;
-
high NK > 40%.
- high NK > 40%. |  |
•
Middle NK – medium individual lysis capacity- people who have
between 25% and 39% natural killer cells lysis capacity;
•
High NK- high individual lysis capacity- people who have over 40%
natural killer cells lysis capacity.
Individual lysis capacity recorded in the
athletes included in the tests
| Individual lysis capacity | Number of cases in judo | Number of cases in
endurance athletes | Number
of cases in speed athletes |
| < 24% | 5 | - | 1 |
| 25% - 29% | 5 | - | 2 |
| 30% - 39% | 4 | 2 | 2 |
| >40% | 1 | 5 | 4 |
 | Picture 2
Individual
lysis capacity recorded in athletics.
Legend: the ordinate
shows the number of cases recorded; the abscissa shows the values for:
-
low NK < 25%,
- middle NK 25% - 29% and 30%-39%;
-
high NK > 40%. |
In
athletics we notice the mirrored distribution of the number of cases
and this would allow for possible explanations for the
already-mentioned situations (sa stii ca nu e foarte clar care sunt
aceste situatii deja discutate):
- The
immune basal status seems to be superior in the athletics group
compared to judo, which makes for the tolerance of an increased effort,
probably existing as a criteria of natural selection;
- It
is not excluded that this immune status might be an effect of the
physical effort itself – this statement is difficult to prove
but also difficult to oppose as long as the primary values from 11-12
years ago are not available;
- The data gives room
for an interesting theme of study: the outlined phenomena are caused
only by the neural-endocrine mechanisms, or are there factors in the
plasma that trigger immune depression and which seem to be activated
only within the hemodynamic modifications caused by effort?
